Sperm are obligate fructose consumers, so there's that. But your body can manufacture fructose as necessary, so it's non-essential in terms of diet.
I have searched in vain for good links to provide regarding how we make fructose. Having failed, I'll simply do my best to elaborate.
Human de novo fructose synthesis is a two-step process involving sorbitol as an intermediary between glucose and fructose, as follows:
aldose reductase sorbitol dehydrogenase
glucose ------------------------> sorbitol ------------------------------------> fructose
The first step of this reaction is the primary pathogenic process of diabetes, where glucose is irreversibly converted to sorbitol in tissues containing aldose reductase: lens, retina, Schwann cells, liver, kidney, placenta, RBCs, ovaries, and seminal vesicles.
The reason this is problematic is that very few tissues contain the sorbitol dehydrogenase enzyme necessary to convert sorbitol to fructose. These tissues are liver, ovaries, and seminal vesicles: the only tissues listed above that have no diabetic pathology. In liver, this process provides a way for sorbitol to enter the glycolytic or gluconeogenic pathways. In the seminal vesicles, it provides fuel for sperm motility. The function is not clear in ovaries.
The reason this first step is problematic in diabetes is the inability of sorbitol to effectively leave the cytoplasm of cells: this results in an altered osmotic gradient, swelling, and loss of function (cataracts, peripheral neuropathy, vascular damage, et al.).