Not to burst anybody's bubble but there can be statistical fluctuations. In my experience, a change within +/- 0.50 in one or both eyes is common from year to year because the refraction for glasses is subjective - and even objective measurements like an autorefractor can have more fluctuation then that! You could have been slightly overcorrected (sometimes by an entire 1.00 diopter in each eye) in the past. Sometimes when people change eye doctors, this is corrected
I tried eye exercises and those books that claim to get rid of glasses when I hit puberty and got glasses (low prescription that time). It didn't work for me. I was a bookworm devouring 1 novel per day(Nancy Drew, Hardy Boys, etc.) so looking back I'm sure that contributed to my myopia.
I did do some VT - vision therapy and vision training to help with eyestrain at near/computer work which did help subjectively and objectively (measured with prisms). My mother did the same. But neither of us eliminated our glasses.
There are some genes involved with myopia. I do believe in epigenetics - gene interaction with the environment so I don't think everyone is doomed by their genes. I would also like to see someone cured without refractive surgery (LASIK, and the like) from -3.00 D to 0 (no prescription) in their glasses.
That being said, it seems that Vitamin D and being outdoors affects myopia!
Now high insulin levels can affect myopia. There are other issues that high-carb, glucose, and insulin negatively affect the eye.
Illiterate and less-educated populations have much lower myopia rates vs. more educated and literate populations as shown in China (rural vs. urban) and Israel (Orthodox vs. Reform Jews - the former have to memorize and read a lot more of their holy texts). This is even in populations that share much of the same genes, suggesting the environment of constant near work (computer and reading which was never part of our evolutionary history) and perhaps less sunlight/Vitamin D contributes to myopia.
I think it maybe easier to prevent (especially before the age 18) myopia then to change it afterwards. However, I could happily be proven wrong - track your glasses prescriptions!
Frankly, I have yet to see objective data for refractive error change (myopia, presbyopia, and hyperopia) for the Bates method (eye exercises), so I'm a bit skeptical. Objective data would include corneal topography, axial length changes.
The section on claimed success is all I can support because of the lack of objective data:
" As evidence for the effectiveness of the Bates method, proponents point to the many accounts of people allegedly having improved their eyesight by applying it. While these anecdotes may be told and passed on in good faith, several potential explanations exist for the phenomena reported other than a genuine reversal of a refractive error due to the techniques practiced:
Some cases of nearsightedness are recognized as due to a transient spasm of the ciliary muscle, rather than a misshapen eyeball. These are classed as pseudomyopia, of which spontaneous reversal may account for some reports of improvement.
Research has confirmed that when nearsighted subjects remove their corrective lenses, over time there is a limited improvement (termed "blur adaptation") in their unaided visual resolution, even though autorefraction indicates no corresponding change in refractive error. This is believed to occur due to adjustments made in the visual system. One who has been practicing Bates' techniques and notices such improvement may not realize that simply leaving the glasses off would have had the same effect, which may be especially pronounced if the prescription was too strong to begin with.
Visual acuity is affected by the size of the pupil. When it constricts (such as in response to an increase in light), the quality of focus will improve significantly, at the cost of a reduced ability to see in dim light. This is known as the "pinhole effect".
Some eye defects may naturally change for the better with age or in cycles (ophthalmologist Stewart Duke-Elder suggested that this is what happened with Aldous Huxley).
A cataract when first setting in sometimes results in much improved eyesight for a short time. One who happens to have been practicing the Bates method will likely credit it for any improvement experienced regardless of the actual cause.
Some studies have suggested that a learned ability to interpret blurred images may account for perceived improvements in eyesight. Ophthalmologist Walter B. Lancaster had this to say: "Since seeing is only partly a matter of the image on the retina and the sensation it produces, but is in still larger part a matter of the cerebral processes of synthesis, in which memories play a principal role, it follows that by repetition, by practice, by exercises, one builds up a substratum of memories useful for the interpretation of sensations and facilitates the syntheses which are the major part of seeing." Lancaster faulted ophthalmologists in general for neglecting the role of the brain in the process of seeing, "leaving to irregular, half-trained workers the cultivation of that field".
"Flashes of clear vision"
Bates method enthusiasts often report experiencing "flashes" of clear vision, in which eyesight momentarily becomes much sharper, but then reverts back to its previous state. Such flashes are not the result of squinting, and can occur in one eye at a time or in both eyes at once. Observation has suggested that both the quality and duration of such flashes can be increased with practice, with some subjects holding a substantial improvement for several minutes. Tests of such subjects have found that the temporary improvement in visual acuity is real, but per retinoscopy is not due to any change in refractive error. A 1982 study concluded that such occurrences are best explained as a contact lens-like effect of moisture on the eye, based on increased tear action exhibited by 15 out of 17 subjects who experienced such improvement. A more recent series of studies have proposed that such flashes may be caused by "negative accommodation" (i.e. an active flattening of the lens by the ciliary muscles).